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@DŸHµ1ÂüéìKûoNÃÝ@#Ââ�aÆžÝ/ÌÒÛç@#ÇÇÂ�ùtXß…=ºš$/(€x {W•‡<(¦‰¢€=Þ45­ELÑöÐvØÛ CM}žúÛ (£<þê ¼¼è¢«0¶Ú­­S‘WéÏî¥ìá§(¨Ðx (¨@>w·+Ñê¹·‡*(¬ŒÑcŸ…¨¢£©4QPÀZ_Ζ–÷ÑE#ÃÏ—�#kéE@¹‹ûêBܼ(¢Œ§[qç~4iaE3ãŠ(9û)‹XÛ�P K›øS¨¢€�ô51~|h¢ 1wÝ8:_ß|Î?GúýUýW¿•|Ûþçôü¦N�÷t!êß…ìvíþQE^Ïÿé×þÅÿ™wøVÿ¸�MÿiºËû–Ïïþa:ž;vz?]}îÒÞ4Q^/Õ¿óóÿþšžŸÓÿððÿ ‹ÿúImÿצûp¿]öýÕÛ;mêøøZÚßÝEÊ¿/‘µüÌò½¿þŠ ¾‹ñÙø/ç¶ÕªŠ+è× ÿ üÿ3ó‰ n/ãNŠ*†Hòµ¸r¤o§ê¿�¢ŠCבãE @s¿ fÿuP[‹p¥­ÍPº_áýT�¿¾Š(ñ4i¯ÝE^v¢Š( òá÷R>^ûQEŬ-÷RkiEL�¨×vœ9ÑECçR<¨¢€~«QEÿÙèJé(€àà€ ò¬/È 0ÒÕä·DArialNew ˆ¹ˆ¹ø&»¸ð·ç0¸z[ 0¸"·D‹[SOalNew ˆ¹ˆ¹ø&»¸ð·ç0¸z[ 0¸† ·DTimes New Roman¸ð·ç0¸z[ 0¸¤€@¥ .©  @£nÿý?" dÿd@ÿÿïÿÿ  @@``€€ dð\ð˜D#    /ðXRð$ópVAjvñ é¢`…£ºÿŒGRð$Òäi·J{ákªŽ£{%4ÿ³ŒGƒ ð0�ƒ†A¿ÀÅAÿñÿ@ñ÷ðó€ÐÓº“°öÊš;­”ÇÊš;úgþý4[d[d ¸z[ 0¸ÿÿÿÎþÿÿpûppû@ <ý4ddddL¸ß 0ˆ¹Ð&»ÿ ˆØŠк___PPT10‹°Ö˜·D‹[SOes New ˆ¹ˆ¹¤·Œ·ç0¤·z[ 0œ·†·DTimes New Roman¤·Œ·ç0¤·z[ 0œ· pµpµðë óŸ @Treatment of Parkinson s diseaseŸª óŸ¨Overview of CNSŸ¨¤Act presynaptically by influencing the production, storage, or termination of action of neurotransmitters. Other agents may activate or block postsynaptic receptor.ª’óŸ¨Neurotransmission in the CNSŸ¨ÞSimilar to autonomic nervous system The circuitry of the CNS is much more complex than the autonomic nervous system Contains powerful networks of inhibitory neurons that are constantly active More than 10 neurotransmitters¡&ßtÿþQóŸ¨Synaptic potentialsŸ¨´Receptors at most synapses are coupled to ion channels. Depolarization or hyperpolarization of the postsynaptic membrane, depending on the specific ions that move and the direction¡&µ<ÿþZªJZóŸ¨Excitatory pathwaysŸ¨%Excitatory postsynaptic potential(EPSP) are generated by the following 1. stimulation of an excitatory neuron causes the release of neurotransmitter molecules 2. The influx of Na+ causes a weak depolarization 3. Pass a threshold, and an all-or-none action potential is generated. ¡"HZÞZ&ªóŸ¨Inhibitory pathways Ÿ œResults in a hyperpolarization of the postsynaptic membrane. 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Other agents may activate or block postsynaptic receptor.ª’óŸ¨Neurotransmission in the CNSŸ¨ÞSimilar to autonomic nervous system The circuitry of the CNS is much more complex than the autonomic nervous system Contains powerful networks of inhibitory neurons that are constantly active More than 10 neurotransmitters¡&ßtÿþQóŸ¨Synaptic potentialsŸ¨´Receptors at most synapses are coupled to ion channels. Depolarization or hyperpolarization of the postsynaptic membrane, depending on the specific ions that move and the direction¡&µ<ÿþZªJZóŸ¨Excitatory pathwaysŸ¨%Excitatory postsynaptic potential(EPSP) are generated by the following 1. stimulation of an excitatory neuron causes the release of neurotransmitter molecules 2. The influx of Na+ causes a weak depolarization 3. Pass a threshold, and an all-or-none action potential is generated. ¡"HZÞZ&ªóŸ¨Inhibitory pathways Ÿ œResults in a hyperpolarization of the postsynaptic membrane. Inhibitory postsynaptic potentials (IPSP) are 1. releases neurons releases neurotransmitter molecules, such as ³-aminobutyric acid (GABA) or glycine. Increase in the permeability of specific ions, such as, potassium and chloride ions 2. the influx of chloride and efflux of potassium cause a weak hyperpolarization or IPSP that moves the postsynaptic potential away from its firing threshold. ¡8lcoA"ªt ’ ™Oó Ÿ >Overview of parkinson s diseaseª  Ÿ¨ÿParkinson's disease (PD) is a progressive disorder of the nervous system. With an annual incidence of approximately 20 new cases per 100,000 people. PD is generally age-specific; it is estimated that approximately 1% of the population over age 60 has PD. ªÿó ó Ÿ¨EtiologyŸ¨~Is correlated with a reduction in the activity of inhibitory dopaminergic neurons in the substantia nigra and corpus striatum.ª>=  ó  ó  ŸŸ¨¬Substantia nigra striatum Fire tonically, rather than in response to specific muscular movements or sensory input. GABA dopamine ¡>€­ª6  xó Ÿ¨ etiology( in summary) ªŸ¨ŠDestruction of cells in the substantia nigra results in the degeneration of neurons responsible for secreting dopamine in the neostriatum.ª> R ó Ÿ¨Strategy of treatmentŸ¨zTherapy is aimed at restoring dopamine in the basal ganglia and antogonizing the excitatory effect of cholinergic neurons.ª@ /ó Ÿ¨Drugs used in PDŸ¨ levodopa(L-dopa) and carbidopa ª$ óŸ¨Mechanism of actionŸ¨àLevodopa: replenish the dopamine deficiency. Dopamine itself does not cross the blood-brain barrier. Levodopa is readily transported into the CNS and is coverted to dopamine in the brain. But side effects in the periphery.ª6_,>óŸ¨ Carbidopaª Ÿ¨›A dopamine decarboxylase inhibitor that does not cross the blood-brain barrier Diminishes the metabolism of levodopa in the GI tract and peripheral tissuesª, T(óŸ¨ActionsŸ¨CLevodopa decreases the rigidity, tremors, and other symptoms of PD ª<óŸ¨Therapeutic usesŸ¨BLevodopa in combination with carbidopa is a potent and efficaciousª$ óŸ¨Absorption and metabolismŸ¨oLevodopa has extremely short half-life(1 to 2 hours) Taken on an empty stomach, typically 45 min before a meal.ªhóŸ¨Adverse effectsŸ¨@Peripheral effects: anorexia, nausea, and vomitting Hypotensionª* óŸ¨ CNS effectsŸ¨HVisual and auditory hallucinations and dyskinesia Depression and anxietyª' óŸ¨ Interactions Ÿ¨jThe vitamin pyridoxine(V6) increases the peripheral breakdown of levodopa and diminishes its effectiveness¡&kÿþQªA"óŸ¨ Bromocriptineª Ÿ¨`Is a dopamine receptor agonist Produces little response in patients who do not react to levodopaªX êî$ï € ”ðŒ`ðPð$ð( ð ðPðr ðP S ð€Ä]ø¿ÿð­ `}ðà  ø ð žðr ðP S ð€h_ø¿ÿðð `ðà ø ð žðH ðP ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë.*rÇÕÄî$ï € ”ðŒ ð`ð$ð( ð ð`ðr ð` S ð€<~ø¿ÿð­ `}ðà  ø ð žðr ð` S ð€Ð¦ø¿ÿðð `ðà ø ð žðH ð` ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë.,rǰùjî$ï € ”ðŒ€ðXð$ð( ð ðXðr ðX S ð€Ümø¿ÿð­ `}ðà  ø ð žðr ðX S ð€ä펿ÿðð `ðà Ž ð žðH ðX ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë.+rÇ�½Z î$ï € ”ðŒ°ðdð$ð( ð ðdðr ðd S ð€Øsø¿ÿð­ `}ðà  ø ð žðr ðd S ð€x½ø¿ÿðð `ðà ø ð žðH ðd ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë.,rÇ dß\î$ï € ”ðŒÀðhð$ð( ð ðhðr ðh S ð€twø¿ÿð­ `}ðà  ø ð žðr ðh S ð€P%ø¿ÿðð `ðà ø ð žðH ðh ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë.-rÇPÁ¶î$ï € ”ðŒÐðlð$ð( ð ðlðr ðl S ð€DÕø¿ÿð­ `}ðà  ø ð žðr ðl S ð€Ð±ø¿ÿðð `ðà ø ð žðH ðl ƒ ð0�ƒ“ŽŸ‹”Þ½h¿ÿ ?ð ÿÿÿ€€€»àã33™™™™Ìˆ8Š0º___PPT10‹ë..rÇÀþî$ï € ”ðŒàðpð$ð( ð ðpðr ðp S ð€� ò¿ÿð­ `}ðà  ò ð   !"#$%&'()*+,-./0123456789:;<=>?@ABCDEFGHIJKLMNOPQRSTUVWXYZ[\]^_`abcdefghijklmnopqrstuvwxyz{|}þÿÿÿ€�‚ƒ„…†‡ˆ‰Š‹Œ�Ž��‘’“”•–—´Ëš›œ�žŸ ¡¢£¤¥¦§¨©ª«¬­®¯Äýÿÿÿýÿÿÿ³þÿÿÿµ¶·¸¹º»¼½¾¿™þÿÿÿÂÃ˜ÅÆÇÈÉÊþÿÿÿþÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿRoot Entryÿÿÿÿÿÿÿÿ��d›Oφꪹ)è†ø0rÇÁ Picturesÿÿÿÿÿÿÿÿ ûCurrent Userÿÿÿÿÿÿÿÿÿÿÿÿ)SummaryInformation(ÿÿÿÿ�PowerPoint Document(ÿÿÿÿÿÿÿÿÿÿÿÿ~€†DocumentSummaryInformation8ÿÿÿÿÿÿÿÿÔÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿÿ‹[SOalNew ˆ¹ˆ¹ø&»¸ð·ç0¸z[ 0¸† ·DTimes New Roman¸ð·ç0¸z[ 0¸¤€@¥ .©  @£nÿý?" dÿd@ÿÿïÿÿ  @@``€€ tðlð¨L)     /ðXRð$ópVAjvñ é¢`…£ºÿŒGRð$Òäi·J{ákªŽ£{%4ÿ³ŒGƒ ð0�ƒ†A¿ÀÅAÿñÿ@ñ÷ðó€ÐÓº“°öÊš;­”ÇÊš;úgþý4[d[d ¸z[ 0”ÿÿÿ’þÿÿpûppû@ <ý4ddddL¸ß 0ˆ¹Ð&»ÿ ˆØŠк___PPT10‹°Ö˜·D‹[SOes New ˆ¹ˆ¹¤·Œ·ç0¤·z[ 0œ·†·DTimes New Roman¤·Œ·ç0¤·z[ 0œ· pµpµðWóŸ @Treatment of Parkinson s diseaseŸª óŸ¨Overview of CNSŸ¨¤Act presynaptically by influencing the production, storage, or termination of action of neurotransmitters. Other agents may activate or block postsynaptic receptor.ª’óŸ¨Neurotransmission in the CNSŸ¨ÞSimilar to autonomic nervous system The circuitry of the CNS is much more complex than the autonomic nervous system Contains powerful networks of inhibitory neurons that are constantly active More than 10 neurotransmitters¡&ßtÿþQóŸ¨Synaptic potentialsŸ¨´Receptors at most synapses are coupled to ion channels. Depolarization or hyperpolarization of the postsynaptic membrane, depending on the specific ions that move and the direction¡&µ<ÿþZªJZóŸ¨Excitatory pathwaysŸ¨%Excitatory postsynaptic potential(EPSP) are generated by the following 1. stimulation of an excitatory neuron causes the release of neurotransmitter molecules 2. The influx of Na+ causes a weak depolarization 3. Pass a threshold, and an all-or-none action potential is generated. ¡"HZÞZ&ªóŸ¨Inhibitory pathways Ÿ œResults in a hyperpolarization of the postsynaptic membrane. 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